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This suggests heterogeneity in cardiorenal benefit based on cardiovascular phenotype in SGLT2is, and is an important factor with future research regarding semaglutide’s potential differential effects across CKD populations with or without overt HF[62]

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This resistance to proteolytic degradation has been primarily achieved by alterations to the cleavage site of GLP-1, via substitution of alanine with alternative amino acids (Table 1)